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"Some obese people may make leptin at a greater rate to compensate for a faulty signaling process or action," Friedman says. "If resistance to leptin is partial, rather than complete, more leptin may be required menstrual cycle for action." In addition, obesity may occur if genetic or environmental factors menstrual cycle affect body chemistry after leptin acts, notes lead author Margherita Maffei, Ph.D., postdoctoral associate in Friedman's laboratory. Leptin's signaling ability may also help explain the high rates of regaining weight found among dieters, the investigators report. "After dieting, the levels of leptin drop, suggesting that menstrual cycle less leptin is made and available to signal the brain," Friedman says. "This reduction may contribute to increased hunger and slower metabolism. If this is true, leptin therapy may help people maintain weight loss after dieting. However, we first must continue with studies to determine the safety of leptin as a possible therapy." The researchers previously published a study documenting a 30 percent weight loss in genetically obese mice given daily injections of leptin for two weeks.
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