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| medical news, air displacement plethysmograph, bubble jet, body fat measurer, burn, harvard school of public health, musical instruments, savin, desirable weight, telephones, measuring tape, aspartate aminotransferase, compare prices, hewlettpackard, wang, fatmonitors, taiwanbody fat monitor, exercise, qms, glass creations, | Prof. Walker: "These studies show that excessive GPR40 action can trigger each of the two fitness 2000 stages of the disease. Our results establish GPR40 as an important fitness 2000 link between fitness 2000 obesity and diabetes. This gives us a new tool to combat the diabetes epidemic: For example, it might be possible in the future to treat the condition using drugs that block the action of this receptor." Prof. Michael Walker's research is supported by the Laufer Charitable Trust; Ms. Ellen Rosenthal, Potomac, MD; Mr. & Mrs. Mitchell; and Cynthia Caplan, Bethesda, MD. Prof. Walker is the incumbent of the Marvin Myer & Jenny Cyker Professorial Chair for Diabetes Research. The American Committee for the Weizmann Institute of Science (ACWIS), founded in 1944, develops philanthropic support for the Weizmann Institute of Science in Israel, one of the premier scientific research institutions. |
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| Helena Edlund and post-doctoral fellow Dr. Per Steneberg of the University of Umea. Together, they developed two types of lab mice with modified GPR40 activity. In the wang first, the scientists used a technique known as gene knock-out to prevent production of the GPR40 receptor. The second type had overactive GPR40 genes creating a surfeit of fat-signaling receptors that tricked the beta cells into sensing high fatty acid levels, even on a normal diet. Throughout the trial, the GPR40 knock-out wang mice remained wang healthy, apparently suffering no ill-effects from the deletion of the receptor, even when the fat content of their diet was raised substantially. In contrast, normal mice on a high-fat diet displayed typical symptoms of the first stage of diabetes. But strikingly, in the animals with extra GPR40 receptors, the disease progression was swift: They soon began to exhibit the classic symptoms of full blown diabetes, including failure of the beta cells to produce adequate amounts of insulin. |
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