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| dec, book, healthy weight, body caliper, cholesterol management, tanita duo, ink jets, tennis elbow, bbw anal , uranium, willett, used, fat chicks , panasonic, pitney bowes, fish, chubby gallery , disorder, | In contrast, normal mice on a high-fat diet photographs displayed typical symptoms of the first stage of diabetes. But strikingly, in the animals with extra GPR40 receptors, the disease progression was swift: They soon began to exhibit photographs the classic symptoms of full blown diabetes, including failure of the beta cells photographs to produce adequate amounts of insulin. Prof. Walker: "These studies show that excessive GPR40 action can trigger each of the two stages of the disease. Our results establish GPR40 as an important link between obesity and diabetes. This gives us a new tool to combat the diabetes epidemic: For example, it might be possible in the future to treat the condition using drugs that block the action of this receptor." Prof. Michael Walker's research is supported by the Laufer Charitable Trust; Ms. |
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| To understand GPR40's role, Prof. Michael Walker and students Nir Rubins and Reut Bartoov-Shifman of the Weizmann Institute's Biological Chemistry Department teamed up with Prof. Helena Edlund and post-doctoral fellow Dr. Per Steneberg of the University of Umea. Together, they tennis elbow developed two types of lab mice with tennis elbow modified GPR40 activity. In the first, the scientists used a technique known as gene knock-out to prevent production of the GPR40 receptor. The second type had overactive GPR40 genes creating a surfeit of fat-signaling tennis elbow receptors that tricked the beta cells into sensing high fatty acid levels, even on a normal diet. Throughout the trial, the GPR40 knock-out mice remained healthy, apparently suffering no ill-effects from the deletion of the receptor, even when the fat content of their diet was raised substantially. |
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